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              <text>Susanne Altmeyer&lt;br /&gt;Leonie Wollersheim&lt;br /&gt;Niclas Kilian-Hütten&lt;br /&gt;Alexander Behnke&lt;br /&gt;Arne Hofmann&lt;br /&gt;Visal Tumani</text>
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              <text>Altmeyer, S., Wollersheim, L., Kilian-Hütten, N., Behnke, A., Hofmann, A., &amp;amp; Tumani, V. (2022, May). Effectiveness of treating depression with EMDR among inpatients -a follow-up study over 12 months. Frontiers in Psychology. doi:10.3389/fpsyg.2022.937204</text>
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                <text>Effectiveness of treating depression with EMDR among inpatients -a follow-up study over 12 months</text>
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                <text>Increasing prevalence of depression poses a huge challenge to the health care systems, and the success rates of current standard therapies are limited. While 30% of treated patients do not experience a full remission after treatment, more than 75% of patients suffer from recurrent depressive episodes. EMDR therapy represents an emerging treatment option of depression, and preliminary studies show promising effects with a probably higher remission rate when compared to control-therapies such as cognitive behavioral therapy. In the present study, 49 patients with severe depression were treated with an integrated systemic treatment approach including EMDR therapy that followed a specific protocol with a treatment algorithm for depression in a naturalistic hospital setting. Following their discharge from the hospital, the patients were followed up by a structured telephone interview after 3 and 12 months. 27 of the 49 (55%) patients fulfilled the Beck’s depression criteria of a full remission when they were discharged. At the follow-up interview, 12 months after discharge, 7 of the 27 patients (26%) reported a relapse, while the remaining 20 patients (74 %) had stayed relapse-free. The findings of our observational study confirm reports of earlier studies in patients with depression, showing that EMDR therapy leads to a high rate of remission, and is associated with a decreased number of relapses. Patients with depression receiving EMDR treatment may be more resilient to stressors.</text>
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                <text>Frontiers in Psychology. doi:10.3389/fpsyg.2022.937204</text>
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              <text>Mohammad Behnam Moghadam &lt;br /&gt;Aziz Behnam Moghadam &lt;br /&gt;Tahmineh Salehian</text>
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              <text>Behnam, M. M., Behnam, M. A., &amp;amp; Salehian, T. (2015). &lt;a href="http://www.efpe.fr/telechargement/articles-emdr/Behnammoghadam%202015%20Efficacy%20of%20EMDR%20on%20depression%20in%20patients%20with%20Myoc....pdf"&gt;Efficacy of eye movement desensitization and reprocessing (EMDR) on depression in patients with myocardial infarction (MI) in a 12-month follow up. &lt;/a&gt;Iranian Journal of Critical Care Nursing, 7(4), 221-226.</text>
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                <text>Efficacy of eye movement desensitization and reprocessing (EMDR) on depression in patients with myocardial infarction (MI) in a 12-month follow up</text>
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                <text>Aims: Depression following Myocardial Infarction (MI) is a common disorder with a negative effect on prognosis of cardiac patients. One of the therapeutic methods of depression in cardiac patients is cognitive-behavioral technique. The aim of the present study was to assess the efficacy of Eye Movement Desensitization and Reprocessing (EMDR) on depression in patients with Myocardial Infarction (MI) in a 12-month follow up. &lt;br /&gt;&lt;br /&gt;Methods: It was a quasi-experimental study, which was performed on patients with MI in Qazvin in year 2013. 60 patients, who were suffering from MI were selected through convenient sampling and based on Beck’s depression questionnaire; they were randomly divided into two experimental and control groups. EMDR therapeutic method was performed in three sessions in the experimental group. The control group received no intervention. Data collection on depressive symptoms was done before treatment, after treatment and in a 12-month follow up and they were analyzed by using descriptive statistics, repeated measures ANOVA, chi- square and SPSS 17 software. &lt;br /&gt;&lt;br /&gt;Results: Depressive symptoms mean in the experimental group before and after intervention and in a 12-month follow up was 27.26±6.41, 11.76±3.71 and 8.5±2.52 respectively; repeated measures ANOVA showed significant statistical difference (p&amp;lt;0.001). Depressive symptoms mean in the control group before and after intervention was 24.53±5.81 and 31.66±6.09 respectively, it showed significant difference by using paired t-test (p&amp;lt;0.001). &lt;br /&gt;&lt;br /&gt;Conclusions: The EMDR is an efficient method for treating and reducing depression in patients with MI. Critical care nurses can use this new and effective method for treating depression.</text>
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                <text>Iranian Journal of Critical Care Nursing, 7(4), 221-226</text>
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              <text>Wilson, S., Becker, L., &amp;amp; Tinker, R. H. (1995, June). 15-Month follow up of EMDR treatment for traumatic memory. Presentation at the EMDR Network Conference, Santa Monica, CA</text>
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                <text>&lt;p&gt;We previously reported on the outcomes of a controlled study of eye movement desensitization and reprocessing (EMDR) effectiveness in the treatment of traumatic memory (Wilson, Tinker, &amp;amp; Becker, 1994; Wilson, Becker, &amp;amp; tinker, in press). In that study we found that three, 90-minute sessions of EMDR (Shapiro, 1995) "normalized the psychological functioning of the previously traumatized participants (g = 80) on all dependent measures. The present study is a 15-month follow up of those participants. I Method: The research design is shown in Table 1. Participants were randomly assigned to EMDR or to Delayed EMDR conditions. Pretreatment measurement occurred at measurement time TI. Participants in the EMDR condition received EMDR between T1 and T2; those in the Delayed EMDR condition received EMDR between T2 and T3. All participants were tested immediately following treatment and at 3 months following treatment (at T4). The 15 month, long-term follow up occurred at measurement time T5. An independent assessor collected all of the following dependent measures: Subjective Units of Disturbance Scale (SUDS; Wolpe, 1990), Impact of Events Scale (IES; Hmowitz, Wilner, &amp;amp; Alvarez, 1979), State/Trait Anxiety Inventory (STAI; Spielberger, Gorsuch, Lushene, Vagg, &amp;amp; Jacobs, 1983), and the Symptom Checklist (SCL-90-R, Derogatis, 1992). [Table 1. The Research Design, Treatment Condition, Measurement Time: T1 T2 T3 T4 T5; EMDR Treatment: 01 x 02 03 04; Delayed EMDR Treatment 01 02 x 03 04 05; Note: T = Time of measurement; 0 = Observation; X = Treatment administered.] II. Results: Two analyses were performed to assess the impact of EMDR treatment at the 15-month follow up. First, in order to assess the overall, long-term impact of EMDR, the 15-month follow-up scores were compared with the pretreatment scores. There was significant improvement on all nine measures at the 15-month follow up: The multivariate effect was significant (Wilk's Lambda =.11, p&amp;lt;.0005) as were all nine of the univariate effects (all p &amp;lt;.0005). Second, in order to assess whether the improvement shown immediately following EMDR treatment had been maintained over the following year the immediate posttreatment scores were compared with, the 15-month follow-up scores. The multivariate test was nonsignificant (Wilk's lambda=.74, p=.079), indicating the improvement shown immediately following EMDR was maintained 15 months later. The univariate analyses indicated additional improvement for the PTSD symptoms of intrusions (IES Intrusion: F(1,56)=7.71, p=307) and avoidance (IES avoidance: F_(1,56) -4.44, p=.040). None of the nine measures showed deterioration at the 15-month follow up. Prior to EMDR treatment 45% (g= 9) of the responders had been diagnosed as PTSD, at the 15-month follow up only 7% (g = 4) were diagnosed as PTSD (chi-squareo, N=61)= .72, p &amp;lt; .05). III. Responders Versus Nonresponders at the 15-Month Follow up.: At the time of writing this abstract, 75% of the participants (g=61) have responded to the 15-month follow up. In general, measures taken prior to treatment did not differentiate responders fiom nonresponders. Responding at the 15-month follow up was unrelated to age, gender, marital status or years of education, although the annual income of the responders (Mdn=21,500) was higher than that of the nonresponders (Mdn = 14,750, Mann-Whitney U=372.5, p=.017). Responding or not at 15 months was unrelated to the type of trauma experienced, whether or not the participants had been in therapy prior to EMDR treatment, or how long ago the trauma had occurred. It was also unrelated to the severity of the trauma as measured by the pretreatment scores on the nine dependent variables and to whether or not the participant met the PTSD diagnosis criteria prior to treatment. A multiple regression analysis used the immediate posttreatment and 90-day posttreatment scores to predict whether or not the participant responded at the 15-month follow up. Nonrespondents were more likely to be depressed at 90-days following treatment than were respondents (R square=.O8, B=-.16, Beta = -.28, F_L1,71)=5.99, p=.017). No other variables entered into the regression model. IV Discussiona and Conclusion, Tretement effects found immediately following EMDR treatment wer maintained or improved 15 months later and thee was a significant decrease in the number of participants diagnosed as PTSD at the 15 month follow up. The comparison of responders to nonresponders at the 15 month follow up showed that the nonresponders were more depressed than the responders, raising the possiblity that the present results may be favorably biased to some extent. The discussion will include the additional, subjective impressions of participants who did not respond to the follow up.  Limitations of EMDR with this population will be discussed, including the influence of comorbidity, multiple traumas, retraumatization after treatment, and spontaneous recurrence of symptoms.  V. References: 1) Derogatis, L. R. (1992). SCL-90: Administration Scoring and Procedures Manual II. Baltimore: Clinical Psychometric Research. 2) Horowitz, M. J., Wilmer, N. &amp;amp; Alverez, W. (1979). Impact of Event Scale: A Measure of Subjective Distress. Psychosomatic Medicine, 41, 209-218. 3) Shapiro, F. (1995), Eye Movement Desensitization and Reprocessing: Basic Principles, Protocols, and Procedures. 4) Speilberger, C. D., Gorsuch, R. L., Lushene, R. D., Vagg, P. R., &amp;amp; Jacobs, G. A. (1983). Manual for the State-Trait Anxiety Inventory, Palo Alto: Consulting Psychologists Press. 5) Wilson, S. A., Tinker, R. A., &amp;amp; Becker, L. A. (1994, November).  Efficacy of Eye Movement Desensitization and Reprocessing (EMDR)Treatment for Trauma Victims. Paper presented at the Annual Meeting of the International Society for Traumatic Stress Studies, Chicago, IL.  6) Wilson, S. A., Becker, L. A., &amp;amp; Tinker, R. A. (In press), EMDR, treatment for psychologically traumatized individuals, Journal of Consulting and Clinical Psychology.&lt;/p&gt;</text>
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              <text>Marco Pagani&lt;br /&gt;Goran Hogberg&lt;br /&gt;Dario Salmaso&lt;br /&gt;Davide Nardo&lt;br /&gt;Orjan Sundin&lt;br /&gt;Cathrine Jonsson&lt;br /&gt;Joaquim Soares&lt;br /&gt;Anna Aberg-Wistedt&lt;br /&gt;Hans Jacobsson&lt;br /&gt;Stig. A. Larsson&lt;br /&gt;Tore Hallstrom</text>
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              <text>99mTc-HMPAO Distribution, Posttraumatic Stress Disorder, PTSD</text>
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              <text>Pagani, M., Hogberg, G., Salmaso, D.,  Nardo, D., Sundin, O., Jonsson, C., Soares, J., Aberg-Wistedt, A., Jacobsson, H., Larsson, S. A., &amp;amp; Hallstrom, T. (2007, October). &lt;a href="http://dx.doi.org/10.1097/MNM.0b013e3282742035" target="_blank"&gt;Effects of EMDR psychotherapy on 99mTc-HMPAO distribution in occupation-related post-traumatic stress disorder.&lt;/a&gt; Nuclear Medicine Communications, 28(10), 757-765. doi:10.1097/MNM.0b013e3282742035</text>
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                <text>Effects of EMDR psychotherapy on 99mTc-HMPAO distribution in occupation-related post-traumatic stress disorder</text>
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                <text>Background: Post-traumatic stress disorder (PTSD) is a derangement of mood control with involuntary, emotionally fraught recollections that may follow deep psychological trauma in susceptible individuals. This condition is treated with pharmacological and/or cognitive therapies as well as psychotherapy with eye movement desensitization and reprocessing (EMDR). However, only a very limited number of studies have been published dealing with work-related PTSD, and investigations on the effect of treatment on cerebral blood flow represent an even smaller number. Aim: To investigate the short-term outcome of occupation-related PTSD after EMDR therapy by 99mTc-HMPAO SPECT. Method: Fifteen patients, either train drivers suffering from PTSD after having been unintentionally responsible for a person-under-train accident or employees assaulted in the course of duty, were recruited for the study. 99mTc-HMPAO SPECT was performed on these patients both before and after EMDR therapy while they listened to a script portraying the traumatic event. Tracer distribution analysis was then carried out at volume of interest (VOI) level using a three-dimensional standardized brain atlas, and at voxel level by SPM. The CBF data of the 15 patients were compared before and after treatment as well as with those of a group of 27 controls who had been exposed to the same psychological traumas without developing PTSD. Results: At VOI analysis significant CBF distribution differences were found between controls and patients before and after treatment (P=0.023 and P=0.0039, respectively). Eleven of the 15 patients responded to treatment, i.e., following EMDR they no longer fulfilled the DSM-IV criteria for PTSD. When comparing only the eleven responders with the controls, the significant group difference found before EMDR (P=0.019) disappeared after treatment. Responders and non-responders showed after therapy significant regional differences in frontal, parieto-occipital and visual cortex and in hippocampus. SPM analysis showed significant uptake differences between patients and controls in the orbitofrontal cortex (Brodmann 11) and the temporal pole (Brodmann 38) both before and after treatment. A significant tracer distribution difference present before treatment in the uncus (Brodmann 36) disappeared after treatment, while a significant difference appeared in the lateral temporal lobe (Brodmann 21). Conclusion: Significant 99mTc-HMPAO uptake regional differences were found, mainly in the peri-limbic cortex, between PTSD patients and controls exposed to trauma but not developing PTSD. Tracer uptake differences between responders and patients not responding to EMDR were found after treatment suggesting a trend towards normalization of tracer distribution after successful therapy. These findings in occupational related PTSD are consistent with previously described effects of psychotherapy on anxiety disorders. [PubMed]</text>
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